Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells

Research output: Contribution to journal › Journal article › Research › peer-review

Standard

Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells. / Hansen, Peter Riis; Rieneck, Klaus; Bendtzen, Klaus.

In: Immunology Letters, Vol. 91, No. 2-3, 15.02.2004, p. 87-91.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Hansen, PR, Rieneck, K & Bendtzen, K 2004, 'Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells', Immunology Letters, vol. 91, no. 2-3, pp. 87-91. https://doi.org/10.1016/j.imlet.2003.11.008

APA

Hansen, P. R., Rieneck, K., & Bendtzen, K. (2004). Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells. Immunology Letters, 91(2-3), 87-91. https://doi.org/10.1016/j.imlet.2003.11.008

Vancouver

Hansen PR, Rieneck K, Bendtzen K. Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells. Immunology Letters. 2004 Feb 15;91(2-3):87-91. https://doi.org/10.1016/j.imlet.2003.11.008

Author

Hansen, Peter Riis ; Rieneck, Klaus ; Bendtzen, Klaus. / Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells. In: Immunology Letters. 2004 ; Vol. 91, No. 2-3. pp. 87-91.

Bibtex

@article{17718be73a9e4630be4ce9961a225319,
title = "Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells",
abstract = "The mineralocorticoid receptor antagonist spironolactone (SPIR) reduces the mortality and morbidity in patients with congestive heart failure (CHF). Overexpression of proinflammatory cytokines contribute to the development and progression of CHF.",
author = "Hansen, {Peter Riis} and Klaus Rieneck and Klaus Bendtzen",
year = "2004",
month = feb,
day = "15",
doi = "http://dx.doi.org/10.1016/j.imlet.2003.11.008",
language = "English",
volume = "91",
pages = "87--91",
journal = "Immunology Letters",
issn = "0165-2478",
publisher = "Elsevier",
number = "2-3",

}

RIS

TY - JOUR

T1 - Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells

AU - Hansen, Peter Riis

AU - Rieneck, Klaus

AU - Bendtzen, Klaus

PY - 2004/2/15

Y1 - 2004/2/15

N2 - The mineralocorticoid receptor antagonist spironolactone (SPIR) reduces the mortality and morbidity in patients with congestive heart failure (CHF). Overexpression of proinflammatory cytokines contribute to the development and progression of CHF.

AB - The mineralocorticoid receptor antagonist spironolactone (SPIR) reduces the mortality and morbidity in patients with congestive heart failure (CHF). Overexpression of proinflammatory cytokines contribute to the development and progression of CHF.

U2 - http://dx.doi.org/10.1016/j.imlet.2003.11.008

DO - http://dx.doi.org/10.1016/j.imlet.2003.11.008

M3 - Journal article

VL - 91

SP - 87

EP - 91

JO - Immunology Letters

JF - Immunology Letters

SN - 0165-2478

IS - 2-3

ER -

ID: 34077034