Morten Frödin
Associate Professor
Frödin Group
Ole Maaløes Vej 5, 2200 København N.
Member of:
ORCID: 0000-0003-3691-9820
1 - 3 out of 3Page size: 100
- 2017
- Published
CRISPR/Cas9 Engineering of Adult Mouse Liver Demonstrates That the Dnajb1–Prkaca Gene Fusion Is Sufficient to Induce Tumors Resembling Fibrolamellar Hepatocellular Carcinoma
Engelholm, Lars Henning, Riaz, A., Serra, Denise, Dagnæs-Hansen, F., Johansen, Jens Vilstrup, Santoni Rugiu, Eric, Hansen, S. H., Niola, Francesco & Frödin, Morten, 1 Dec 2017, In: Gastroenterology. 153, 6, p. 1662-1673.e10Research output: Contribution to journal › Journal article › Research › peer-review
- Published
The focal adhesion-associated proteins DOCK5 and GIT2 comprise a rheostat in control of epithelial invasion
Frank, S. R., Köllmann, C. P., van Lidth de Jeude, J. F., Thiagarajah, J. R., Engelholm, Lars Henning, Frödin, Morten & Hansen, S. H., 30 Mar 2017, In: Oncogene. 36, 13, p. 1816-1828 13 p.Research output: Contribution to journal › Journal article › Research › peer-review
- Published
Genome editing using FACS enrichment of nuclease-expressing cells and indel detection by amplicon analysis
Lonowski, Lindsey Allison, Narimatsu, Yoshiki, Riaz, A., Delay, C. M. E., Yang, Zhang, Niola, Francesco, Duda, K., Ober, Elke, Clausen, Henrik, Wandall, Hans H., Hansen, S. H., Bennett, Eric Paul & Frödin, Morten, Mar 2017, In: Nature Protocols. 12, 3, p. 581-603 23 p.Research output: Contribution to journal › Journal article › Research › peer-review
ID: 9576
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CRISPR/Cas9 Engineering of Adult Mouse Liver Demonstrates That the Dnajb1–Prkaca Gene Fusion Is Sufficient to Induce Tumors Resembling Fibrolamellar Hepatocellular Carcinoma
Research output: Contribution to journal › Journal article › Research › peer-review
Published -
176
downloads
The focal adhesion-associated proteins DOCK5 and GIT2 comprise a rheostat in control of epithelial invasion
Research output: Contribution to journal › Journal article › Research › peer-review
Published -
100
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p190 RhoGAP promotes contact inhibition in epithelial cells by repressing YAP activity
Research output: Contribution to journal › Journal article › Research › peer-review
Published