Michal Tomasz Marzec
Guest researcher
Inflammation, Metabolism and Oxidation
Blegdamsvej 3
2200 København N.
ORCID: 0000-0001-9698-6019
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Inhibition of ALK enzymatic activity in T-cell lymphoma cells induces apoptosis and suppresses proliferation and STAT3 phosphorylation independently of Jak3
Marzec, Michal Tomasz, Kasprzycka, M., Ptasznik, A., Wlodarski, P., Zhang, Q., Ødum, Niels & Wasik, M. A., 2005, In: Laboratory Investigation. 85, 12, p. 1544-54 10 p.Research output: Contribution to journal › Journal article › Research › peer-review
Activation of Mammalian Target of Rapamycin in Transformed B Lymphocytes Is Nutrient Dependent but Independent of Akt, Mitogen-Activated Protein Kinase/Extracellular Signal-Regulated Kinase Kinase, Insulin Growth Factor-I, and Serum.
Wlodarski, P., Kasprzycka, M., Liu, X., Marzec, Michal Tomasz, Robertson, E. S., Slupianek, A. & Wasik, M. A., 2005, In: Cancer Research. 65, 17, p. 7800-7808 9 p.Research output: Contribution to journal › Journal article › Research › peer-review
STAT3- and DNA methyltransferase 1-mediated epigenetic silencing of SHP-1 tyrosine phosphatase tumor suppressor gene in malignant T lymphocytes.
Zhang, Q., Wang, H. Y., Marzec, Michal Tomasz, Raghunath, P. N., Nagasawa, T. & Wasik, M. A., 2005, In: Proceedings of the National Academy of Sciences of the United States of America. 102, 19, p. 6948-6953 6 p.Research output: Contribution to journal › Journal article › Research › peer-review
ID: 129941679
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134
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Simultaneous inhibition of mTOR-containing complex 1 (mTORC1) and MNK induces apoptosis of cutaneous T-cell lymphoma (CTCL) cells
Research output: Contribution to journal › Journal article › Research › peer-review
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119
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Glutathione S-Transferase P1 Genetic Variant’s Influence on the HbA1c Level in Type Two Diabetic Patients
Research output: Contribution to journal › Journal article › Research › peer-review
Published -
90
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The intermediate proteasome is constitutively expressed in pancreatic beta cells and upregulated by stimulatory, low concentrations of interleukin 1 β
Research output: Contribution to journal › Journal article › Research › peer-review
Published