Michal Tomasz Marzec

Michal Tomasz Marzec

Guest researcher

ORCID: 0000-0001-9698-6019

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  1. Prediction and Early Detection of Response by NMR Spectroscopy and Imaging

    Lee, S., Arias-Mendoza, F., Poptani, H., Delikatny, E. J., Wasik, M., Marzec, Michal Tomasz, Schuster, S. J., Nasta, S. D., Svoboda, J., O'Connor, O. A., Smith, M. R. & Glickson, J. D., Jan 2012, In: PET Clinics. 7, 1, p. 119-26 8 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  2. Plasma gelsolin modulates cellular response to sphingosine 1-phosphate

    Bucki, R., Kulakowska, A., Byfield, F. J., Zendzian-Piotrowska, M., Baranowski, M., Marzec, Michal Tomasz, Winer, J. P., Ciccarelli, N. J., Gorski, J., Drozdowski, W., Bittman, R. & Janmey, P. A., 2010, In: American Journal of Physiology: Cell Physiology. 299, 6, p. C1516-23

    Research output: Contribution to journal › Journal article › Research › peer-review

  3. Peroxisome Proliferator-Activated Receptor γ Promotes Lymphocyte Survival through Its Actions on Cellular Metabolic Activities.

    Jo, S., Yang, C., Miao, Q., Marzec, Michal Tomasz, Wasik, M. A., Lu, P. & Wang, Y. L., 2006, In: Journal of Immunology. 177, 6, p. 3737-3745 9 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  4. Published

    Peanut-shaped gold nanoparticles with shells of ceragenin csa-131 display the ability to inhibit ovarian cancer growth in vitro and in a tumor xenograft model

    Piktel, E., Oscilowska, I., Suprewicz, Ł., Depciuch, J., Marcińczyk, N., Chabielska, E., Wolak, P., Głuszek, K., Klimek, J., Zieliński, P. M., Marzec, Michal Tomasz, Savage, P. B., Parlińska-Wojtan, M. & Bucki, R., 2021, In: Cancers. 13, 21, p. 1-26 5424.

    Research output: Contribution to journal › Journal article › Research › peer-review

  5. PU.1 activates transcription of SHP-1 gene in hematopoietic cells.

    Wlodarski, P., Zhang, Q., Liu, X., Kasprzycka, M., Marzec, Michal Tomasz & Wasik, M. A., 2007, In: Journal of Biological Chemistry. 282, 9, p. 6316-6323 8 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  6. Oncogenic tyrosine kinase NPM/ALK induces activation of the rapamycin-sensitive mTOR signaling pathway.

    Marzec, Michal Tomasz, Kasprzycka, M., Liu, X., El-Salem, M., Halasa, K., Raghunath, P. N., Bucki, R., Wlodarski, P. & Wasik, M. A., 2007, In: Oncogene. 26, 38, p. 5606-5614 9 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  7. Oncogenic tyrosine kinase NPM/ALK induces activation of the MEK/ERK signaling pathway independently of c-Raf.

    Marzec, Michal Tomasz, Kasprzycka, M., Liu, X., Raghunath, P. N., Wlodarski, P. & Wasik, M. A., 2007, In: Oncogene. 26, 6, p. 813-821 9 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  8. Oncogenic kinase NPM/ALK induces through STAT3 expression of immunosuppressive protein CD274 (PD-L1, B7-H1)

    Marzec, Michal Tomasz, Zhang, Q., Goradia, A., Raghunath, P. N., Liu, X., Paessler, M., Wang, H. Y., Wysocka, M., Cheng, M., Ruggeri, B. A. & Wasik, M. A., 30 Dec 2008, In: Proceedings of the National Academy of Sciences of the United States of America. 105, 52, p. 20852-7 6 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  9. Published

    Oncogenic kinase NPM/ALK induces expression of HIF1 alpha mRNA

    Marzec, Michal Tomasz, Liu, X., Wong, W., Yang, Y., Pasha, T., Kantekure, K., Zhang, P., Woetmann, Anders , Cheng, M., Ødum, Niels & Wasik, M. A., 2011, In: Oncogene. 30, 11, p. 1372-1378 7 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  10. Nucleophosmin/anaplastic lymphoma kinase (NPM/ALK) oncoprotein induces the T regulatory cell phenotype by activating STAT3

    Kasprzycka, M., Marzec, Michal Tomasz, Liu, X., Zhang, Q. & Wasik, M. A., 27 Jun 2006, In: Proceedings of the National Academy of Sciences of the United States of America. 103, 26, p. 9964-9969

    Research output: Contribution to journal › Journal article › Research › peer-review

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