Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen

Research output: Contribution to journal › Journal article › Research › peer-review

Standard

Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen. / Broom, Oliver Jay; Nielsen, Ole Haagen.

In: Ugeskrift for læger, Vol. 171, No. 4, 2009, p. 243-6.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Broom, OJ & Nielsen, OH 2009, 'Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen', Ugeskrift for læger, vol. 171, no. 4, pp. 243-6.

APA

Broom, O. J., & Nielsen, O. H. (2009). Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen. Ugeskrift for læger, 171(4), 243-6.

Vancouver

Broom OJ, Nielsen OH. Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen. Ugeskrift for læger. 2009;171(4):243-6.

Author

Broom, Oliver Jay ; Nielsen, Ole Haagen. / Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen. In: Ugeskrift for læger. 2009 ; Vol. 171, No. 4. pp. 243-6.

Bibtex

@article{bbd00970373211df8ed1000ea68e967b,
title = "Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen",
abstract = "It has recently been demonstrated that cysteinyl leukotrienes are involved in a variety of proinflammatory and neoplastic functions. This article gives an up-to-date overview of the present knowledge of these bioactive lipid molecules. Inflammatory bowel disease (IBD) and colorectal cancer (CRC) are applied as models of their cellular actions. It is described how signalling cascades initiated by cysteinyl leukotrienes represent alternative ways in which IBD might progress and lead to the development and propagation of CRC, and as such represent potential targets for future therapeutic regimens to manage chronic inflammatory disorders and cancer. Udgivelsesdato: 2009-Jan-19",
author = "Broom, {Oliver Jay} and Nielsen, {Ole Haagen}",
note = "Keywords: Cell Membrane; Cell Proliferation; Cell Transformation, Neoplastic; Colorectal Neoplasms; Humans; Inflammation Mediators; Inflammatory Bowel Diseases; Leukotriene D4; Leukotrienes; Receptors, Leukotriene; Signal Transduction",
year = "2009",
language = "Dansk",
volume = "171",
pages = "243--6",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "4",

}

RIS

TY - JOUR

T1 - Cysteinylleukotrieners betydning for kronisk inflammation og neoplasi i tarmen

AU - Broom, Oliver Jay

AU - Nielsen, Ole Haagen

N1 - Keywords: Cell Membrane; Cell Proliferation; Cell Transformation, Neoplastic; Colorectal Neoplasms; Humans; Inflammation Mediators; Inflammatory Bowel Diseases; Leukotriene D4; Leukotrienes; Receptors, Leukotriene; Signal Transduction

PY - 2009

Y1 - 2009

N2 - It has recently been demonstrated that cysteinyl leukotrienes are involved in a variety of proinflammatory and neoplastic functions. This article gives an up-to-date overview of the present knowledge of these bioactive lipid molecules. Inflammatory bowel disease (IBD) and colorectal cancer (CRC) are applied as models of their cellular actions. It is described how signalling cascades initiated by cysteinyl leukotrienes represent alternative ways in which IBD might progress and lead to the development and propagation of CRC, and as such represent potential targets for future therapeutic regimens to manage chronic inflammatory disorders and cancer. Udgivelsesdato: 2009-Jan-19

AB - It has recently been demonstrated that cysteinyl leukotrienes are involved in a variety of proinflammatory and neoplastic functions. This article gives an up-to-date overview of the present knowledge of these bioactive lipid molecules. Inflammatory bowel disease (IBD) and colorectal cancer (CRC) are applied as models of their cellular actions. It is described how signalling cascades initiated by cysteinyl leukotrienes represent alternative ways in which IBD might progress and lead to the development and propagation of CRC, and as such represent potential targets for future therapeutic regimens to manage chronic inflammatory disorders and cancer. Udgivelsesdato: 2009-Jan-19

M3 - Tidsskriftartikel

C2 - 19174041

VL - 171

SP - 243

EP - 246

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 4

ER -

ID: 18813266