Morten Bækgaard Thomsen
Associate Professor
ORCID: 0000-0002-2469-6458
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Kv7.1 isoform gradients in the heart: new potential approach to alter repolarization reserve
Thomsen, Morten Bækgaard, Aug 2013, In: Heart rhythm : the official journal of the Heart Rhythm Society. 10, 8, p. 1229-30 2 p.Research output: Contribution to journal › Journal article › Research › peer-review
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Development of heart failure is independent of K+ channel-interacting protein 2 expression
Speerschneider, T., Grubb, Søren, Metoska, A., Olesen, S., Callø, Kirstine & Thomsen, Morten Bækgaard, 1 Dec 2013, In: Journal of Physiology. 591, 23, p. 5923-37 15 p.Research output: Contribution to journal › Journal article › Research › peer-review
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Physiology and analysis of the electrocardiographic T wave in mice
Speerschneider, T. & Thomsen, Morten Bækgaard, Dec 2013, In: Acta Physiologica. 209, 4, p. 262-71 10 p.Research output: Contribution to journal › Journal article › Research › peer-review
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In Vivo Phosphoproteomics Analysis Reveals the Cardiac Targets of β-Adrenergic Receptor Signaling
Lundby, Alicia, Andersen, M. N., Steffensen, A. B., Horn, H., Kelstrup, C. D., Francavilla, C., Jensen, Lars Juhl, Schmitt, Nicole, Thomsen, Morten Bækgaard & Olsen, Jesper Velgaard, 4 Jun 2013, In: Science Signaling. 6, 278, p. rs11 13 p.Research output: Contribution to journal › Journal article › Research › peer-review
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Flattening of the electrocardiographic T-wave is a sign of proarrhythmic risk and a reflection of action potential triangulation
Bhuiyan, T. A., Graff, C., Kanters, Jørgen K., Thomsen, Morten Bækgaard & Struijk, J. J., 1 Jan 2013, Computing in Cardiology. Vol. 40. p. 353-356 4 p.Research output: Chapter in Book/Report/Conference proceeding › Book chapter › Research
ID: 9526984
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Potassium Channel Interacting Protein 2 (KChIP2) is not a transcriptional regulator of cardiac electrical remodeling
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128
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Uremia increases QRS duration after β-adrenergic stimulation in mice
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67
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Accelerated atherosclerosis caused by serum amyloid A response in lungs of ApoE−/− mice
Research output: Contribution to journal › Journal article › Research › peer-review
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