Genomet og diabetes

Research output: Contribution to journalJournal articleResearchpeer-review

Standard

Genomet og diabetes. / Allin, Kristine Højgaard; Hansen, Torben; Pedersen, Oluf Borbye.

In: Ugeskrift for Laeger, Vol. 176, No. 23, V06140337, 10.11.2014, p. 2176-2179.

Research output: Contribution to journalJournal articleResearchpeer-review

Harvard

Allin, KH, Hansen, T & Pedersen, OB 2014, 'Genomet og diabetes', Ugeskrift for Laeger, vol. 176, no. 23, V06140337, pp. 2176-2179.

APA

Allin, K. H., Hansen, T., & Pedersen, O. B. (2014). Genomet og diabetes. Ugeskrift for Laeger, 176(23), 2176-2179. [V06140337].

Vancouver

Allin KH, Hansen T, Pedersen OB. Genomet og diabetes. Ugeskrift for Laeger. 2014 Nov 10;176(23):2176-2179. V06140337.

Author

Allin, Kristine Højgaard ; Hansen, Torben ; Pedersen, Oluf Borbye. / Genomet og diabetes. In: Ugeskrift for Laeger. 2014 ; Vol. 176, No. 23. pp. 2176-2179.

Bibtex

@article{4239705056d54342b9a0db0edee2399b,
title = "Genomet og diabetes",
abstract = "In terms of their genetic architecture monogenic diabetes and type 2 diabetes represent two extremes. Whereas each subtype of monogenic diabetes is caused by one penetrant, rare mutation in a single gene, the genetic susceptibility to type 2 diabetes can be attributed to many low-penetrant variants across the genome. At present, only 10{\%} of the genetic susceptibility to type 2 diabetes can be explained by the hitherto identified 90 genomic loci. Here we briefly review the genetics of monogenic diabetes and type 2 diabetes and outline future directions of research within this field.",
author = "Allin, {Kristine H{\o}jgaard} and Torben Hansen and Pedersen, {Oluf Borbye}",
year = "2014",
month = "11",
day = "10",
language = "Dansk",
volume = "176",
pages = "2176--2179",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "23",

}

RIS

TY - JOUR

T1 - Genomet og diabetes

AU - Allin, Kristine Højgaard

AU - Hansen, Torben

AU - Pedersen, Oluf Borbye

PY - 2014/11/10

Y1 - 2014/11/10

N2 - In terms of their genetic architecture monogenic diabetes and type 2 diabetes represent two extremes. Whereas each subtype of monogenic diabetes is caused by one penetrant, rare mutation in a single gene, the genetic susceptibility to type 2 diabetes can be attributed to many low-penetrant variants across the genome. At present, only 10% of the genetic susceptibility to type 2 diabetes can be explained by the hitherto identified 90 genomic loci. Here we briefly review the genetics of monogenic diabetes and type 2 diabetes and outline future directions of research within this field.

AB - In terms of their genetic architecture monogenic diabetes and type 2 diabetes represent two extremes. Whereas each subtype of monogenic diabetes is caused by one penetrant, rare mutation in a single gene, the genetic susceptibility to type 2 diabetes can be attributed to many low-penetrant variants across the genome. At present, only 10% of the genetic susceptibility to type 2 diabetes can be explained by the hitherto identified 90 genomic loci. Here we briefly review the genetics of monogenic diabetes and type 2 diabetes and outline future directions of research within this field.

M3 - Tidsskriftartikel

VL - 176

SP - 2176

EP - 2179

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 23

M1 - V06140337

ER -

ID: 148552424